February 9, 2016
Iron Toxicity Post #17: Could ‘Folate deficiency’ be an EPI-genetic deficiency of bioavailable copper?

(Formerly #20)

This is a real brain twister.

Kouamou, E, et al. (2013). “[An uncommon etiology of anemia: copper deficiency]”
www.ncbi.nlm.nih.gov/m/pubmed/23906580/

Maybe, just maybe, could all this foments about folate deficiency be an epigenetic deficiency of bioavailable copper that is affecting the metabolic function in our iron-stressed cells?

It does conjure up a very different perspective of Mother Nature vs. methylenetetrahydrofolate reductase (MTHFR).

Please note, B-Vitamins are all biogenic amines and they all do not work until they are ‘kissed’ by ceruloplasmin (Cp).

A votre sante!

MORLEY M. ROBBINS 

For Facebook Discussion:
https://www.facebook.com/groups/MagnesiumAdvocacy/permalink/968470819887654/

You may also like

RCP and copper: a change in philosophy

RCP’s approach to copper has changed over time. In the past you might have seen Morley discourage copper supplementation. Now it’s included in the RCP handbook. Find out why it’s now part of the protocol.