Dr. Ben Edwards; You’re The Cure | November 4, 2019

Dr. Ben Edwards; You’re The Cure | November 4, 2019

In this episode Morley and Dr. Ben reinforce the idea that the very, very root cause of “disease” is metabolic chaos at the cellular level. They stress how American’s have lost their resiliency and resistance to disease because the mitochondria get stuck in a low energy, pro-inflammatory mode. Other highlights are:

  • The lack of bioavailable copper is the number one thing driving this lack of resiliency
  • Dietary iron goes right into mitochondria
  • The mitochondria flips into an “M1” state which is characterized by low energy output and high free radical output. This design was to handle the primary stressors in our ancestors lives: germs and trauma.
  • Once the stressor has been taken care of by the pro-inflammatory free radicals then the mitochondria can flip into “M2” state. In the M2 state, the mitochondria makes lots of energy and very little free radicals so that the cell can heal.
  • “Copper histadine” mimics ceruloplasmin and skinbiology.com makes “serum copper” based off this concept for skin and wound repair. It is possible some this copper can be absorbed into the body and have a beneficial impact on ceruloplamin activity
  • Sugar (especially HFCS), roundup, Ascorbic acid, birth control pills, Antibiotics, hormone replacement therapy all cause the usable copper to be forced out of ceruloplasmin so that it isn’t available to do it’s job of activating oxygen.

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Dr Ruscio | Could Supplementing With Iron Be Harming You? | October 19, 2019

Dr Ruscio | Could Supplementing With Iron Be Harming You? | October 19, 2019

Listen

Morley Robbins met with Dr Ruscio earlier in 2019 and there’s a fantastic podcast available and extensive notes, whereby you can get a deeper understanding of many aspects of the RCP.

They talked about the impact of insufficient copper on the body, including how iron and copper interact, and how that impacts on our body.

Low ferritin isn’t necessarily telling us that we have low iron – it doesn’t give us a full picture. Iron in the blood isn’t the same as iron in the tissue.

Morley shared citations to research he’s read going back to 1976 regarding ferritin in the blood, actually lacking iron, so its use as an iron marker being inappropriate for the context it is so commonly relied upon.

“Is iron insufficiency a total myth? Is copper the answer? Morley Robbins joins us to share his take on what’s really going on with iron and copper in our bodies. ”
They talked about the common misunderstanding on copper deficiency signs and how they are often said to be from iron deficiency – understanding is required of the critical pathways copper is required in so a more complete picture can be seen.

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Jenny | The Root Cause Protocol Helped With My Osteoporosis

Ellen | The Root Cause Protocol Helped With My Body Temperature Regulation

“My body temperature regulation has improved tenfold on the protocol. Two years ago and for many years before that people joke that I was a cold-blooded animal. Temperature regulation was from external environments only. But now be completely up to other people’s ability but it’s not something I feel like I suffer from. I can’t tell you exactly what did it I mean the protocol but I don’t know specifically. It’s hard to get on everything and we don’t always tolerate certain things at first but over time as we add in what we can add in it gets better.”

Ellen

4Health by Anna Sparre | Zinc, Copper, Selenium, the Thyroid and Stress | Episode 221b | September 25, 2019

4Health by Anna Sparre | Zinc, Copper, Selenium, the Thyroid and Stress | Episode 221b | September 25, 2019

Morley has appeared as a guest of Anna Sparre’s 4Health podcast, based in Sweden across a series of podcasts.

In this episode, Morley discusses what could happen to your energy production if you supplement with zinc. He also discusses what happens to your mineral status if you are under chronic stress. Morley and Anna also explore how thyroid issues are connected to copper, selenium and vitamin A.

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