Robin Williams suffered from a Magnesium deficiency?…

Robin Williams suffered from a Magnesium deficiency?…

The world has lost a treasured icon this week (8/11/14) with the shocking departure of Robin Williams (1951-2014).

Few of us alive today have not been graced by his gift for outrageous humor, and yet we have also been moved by his talent as a dramatic actor. Who here didn’t think he deserved the Oscar for his epic performance in “Dead Poets Society?” Despite these gifts, he suffered from “depression,” and is alleged to have taken his own life. How could someone sooooo funny, and so able to keep us laughing, struggle mightily with its polar opposite?
In fact, one of the MAG-pies asked today, “Is it possible that mineral imbalances caused his depression?…”
It was a sincere question, and the answer is both simple and sublimely complex.
The answer in one word, “Absolutely!”
Now let me explain…
It has been well documented, since 1926, that excess Calcium and too little Maggie causes depression. A more recent abstract is noted here. I first learned of this disorienting research and this Calcium/Magnesium dynamic from George & Karen Eby, and their wonderful website. Please know, George is not just some internet blogger. He’s a gifted researcher, who invented the Zinc lozenge, and intimately understands the power of minerals. And after suffering from depression for 13 years — he solved his “Zoloft deficiency” with nothing more than Magnesium Glycinate — in a matter of weeks. Much to his and his psychiatrist’s surprise.
And yes, there was a time when I thought the solution was entirely that simple. Regrettably, the more I study these mineral dynamics, the more the plot thickens… but the theme is still very much focused on these *spark plugs* of life that define and dictate our well-being.
Let’s peel the onion back just another layer. And what we learn is that the Melatonin Pathway is profoundly important for normal levels of BOTH Serotonin and Melatonin:
This pathway is profoundly important…
In fact, there ain’t many pathways that are more compromised than this one across the Globe… It sets the stage for a great deal of despair, dismay, and ultimately, depression.
Please note the four distinct stages of metabolic activity that enables us to go from protein (Tryptophan) >> to our favorite sleep hormone (Melatonin), with a stop-off in between with Serotonin. And please also take stock of the many minerals and vitamins — the one’s we’re supposed to be getting from our daily diet — that support (i.e. ENABLE) these key processes to unfold naturally inside our bodies.
Now, let’s take a closer look and study the BIG BLACK “X’s” off to the side of these reactions… These are where excess, unbound Copper can and does BLOCK the pathway. Fascinating, eh?… And the only glitch that I’m aware of in this otherwise outstanding article/pathway is their failure to highlight the profound negative impact that unbound Copper can have on Vitamin-B6… And for those suffering with sleep issues, look no further for the root cause of your now sandbag eyes.
Excess, unbound Copper has a decided ability to destroy three key nutrients: Maggie (Mg), Ziggie (Zn) and their Buddy (B6). Yes, ALL three… And what many are not fully aware of, especially the mineral denialists we tend to turn to in times of strife, is that inside our bodies, there is a key triangle of interdependence taking place:

  • Zinc is essential for the production of B6…
  • B6 is essential for getting Maggie INSIDE the cell…
  • Maggie is essential for keeping Ziggie inside the cell & body, by managing our “Stress!” Response…

Please know, I have used and/or referred to this chart scores of times… It’s simplicity & clarity to outline this pivotal pathway explains where much of what ails us originates. And yes, it IS all about the lack of minerals that are KEY to the optimal function of these critical enzymes. Truth be known, THAT is the very mechanism of “enzyme deficiency.” And given that Mg, Zn & B6 are also easily LOST TO a wide set of “Stressors!” — this seems to be a far more plausible explanation of the physiology, than the contrived nostrums of conventional practitioners seeking to have us believe the deficiency was inspired by asteroid activity on Mars or something to its equivalent.
And until today, I was satisfied that that article explained this metabolic dysfunction.
However, Fate today brought me to a very thoughtful discussion with a Mom in Essex, UK this morning, who just happened to comment on her 5-yr old son’s reaction to banana’s… His what?… Yes, she raised the banana card — and now it’s time for us all to buckle up our seat belts…
What is important to understand about Edward’s (not his real name…) condition is that he’s dealing with excessive Copper and the lowest level of Zinc I’ve ever witnessed on an HTMA (hair tissue mineral analysis). He was vaccinated as an infant and given Soy-based infant formula. (For those in the know, that’s a wicked combo of toxic metals and uber levels of Copper, and he’s now challenged by a notable Zinc deficiency that is contributing to severe food/chemical sensitivities, rashes over much of his body, and an overall lack of growth — his sister, who’s 2 years younger, is bigger than he is.) To learn more about the pivotal role of Ziggie in our bodies, please read this and this.
OK, enough about Ziggie… what’s the deal with “depression” and our focus on minerals?…
It’s actually quite fascinating…
What I discovered today is that there is an alternate pathway that was offered up by the brilliant sleuthing of Emily Deans, MD . There’s an entirely different pathway for L-Tryptophan to go down…. And as Dr. Deans points out, it is this alternate pathway that sets the stage for increased levels of Kynurenine . Huh? (I know what you’re thinkin’… Magnesium Man! is beginning to lose it, right?…)
As it turns out, excess Estrogen can gum the works of a key enzyme Tryptophan 2,3 Oxidase… And when we learn that this pivotal enzyme  must be activated by Ferrous Iron, the Copper connection becomes even more important. If there isn’t sufficient Ceruloplasmin to bind up BOTH the Copper and the Iron, neither one works. And to further compound this dynamic, most, if not all, of the key Iron proteins (Ceruloplasmin, Ferritin, Hephaestin, Transportin, and Hemoglobin) are each Copper dependent… So, when there’s  a notable Zinc/Copper mismatch like there is with Edward, he can’t break down the Tryptophan in the banana, and the day after, he gets very weepy according to his Mom…. Hmmmm. Weepy… all for a lack of bio-available Copper, proper forms of Iron and ultimately a lack of optimal enzyme activity. (Btw, the WORST thing you can do is take supplemental Iron for those low Iron proteins…)
For those inclined to dig a little deeper, here’s a fascinating overview:

So what happens is that the body slows the production of Serotonin (the 5-HTP “feel good” neurotransmitter on the left side of this chart) and then the body starts to increase the manufacture of Kynurenine (a “feel bad” chemical on the right side…)
And what is key to the proper function of the Tryptophan 2,3 Oxidase?… Well, as noted above, it requires Ferrous Iron to be available. When Copper is in excess, and at the same time “deficient,” it blocks the proper production of Ceruloplasmin, so there’s little chance that the proper form of Iron will be available for this reaction and thus setting the stage for yet another stimulus for “depression.”
So for those looking for a simple way to solve this issue of “depression,” please focus on your Magnesium status. But regrettably, there are many — far too many — that are also dealing with a roiling sea of Copper/Zinc mismatch that is destabilizing proper enzyme activity.
Again, as I’ve declared on the MAG FB page time, and time, again:

  • There is NO such thing as “medical disease…”
  • There is ONLY “metabolic dysfunction”…
  • That is CAUSED by “mineral deficiencies…”

Hopefully, this brief overview begins to emphasize the importance of mineral balance and mineral ratios, particularly as it relates to this concept of “depression” now being discussed ad nauseum on the media, yet with NO explanation for the actual physiological and mineral dynamics driving this set of symptoms.
I will close this post by stating that there is an historical connection between suicide and Mg deficiency. Given that Robin Williams is believed to have taken his own life, this is a relevant nuance to examine.
In 1968, J.I. Rodale wrote a small, but incredibly important book on the vital role of Maggie in our day-to-day lives. By the grace of God, Paul Mason has this amazing book on his website. The ENTIRE book is well worth the read, but Chapter 19 is most relevant to our discussion.
There is, and has been, a clear and compelling link between severe Mg deficiency and the taking of one’s life. In fact, the feelings of “helplessness & hopelessness” that often precede the willingness to take one’s life, are recognized as a profound state of “Stress!” — and this state of mind, too, CAUSES a massive loss of Magnesium.
So, yes, the world has lost one of it’s greatest comics & actors. But it is a tragedy for three distinct reasons:

  • Our world will never laugh, nor be moved in the same way again…
  • The entire medical community is ill-equipped to explain the mineral dynamics outlined above, much less naturally correct the biochemistry of what has been shared… and
  • The entire arsenal of “solutions” offered by our doctors, in fact, CAUSE mineral loss, esp. to Magnesium status.

Please re-read that third bullet, again… I’ll let you grasp the “1984!”-esque quality of this total disconnect (where White is black, & Black is white…) with human physiology that is VERY well chronicled in the literature, but is apparently sparingly read, nor taught in the hallowed halls of medical school classrooms & libraries around the world.
So what are some of the factors that led up to Robin Williams wanting to take his life? A long history of drug abuse, doing cocaine with John Belushi until John’s sudden death, most probably from a severe magnesium loss.  Apparently, Robin also abused alcohol and he couldn’t stay sober even with repeated treatment. Alcohol and stimulant drug abuse BOTH deplete Mg and accelerate our MBR (Mg Burn Rate). It is pure speculation, but I suspect that he had a pretty high MBR as a result of long-term drug and alcohol abuse, known angst about some of the actions that he had taken, ultimately resulting in a significant deficiency of his Magnesium stores.
May God Bless Robin Williams, his entire extended network of family & friends who are grieving his loss, and ALL those individuals around the Globe seeking to liberate themselves from this ever growing bondage of “depression.”
[And for those wondering why my “passion meter” is a bit cranked on this particular blog, it’s worth noting that my Dad suffered from “manic-depression” throughout his entire adult life, until his untimely death at the age of 56. He ultimately died of Lung Cancer, but my Mom shared with me that on more than one occasion, he contemplated committing suicide… What I wouldn’t do to share these simple, but profound insights about HOW & WHY this happened to him — and to each of us  in our family.]
A votre sante!

Ascorbic Acid Causes Copper Deficiency?… Huh?!?…

Ascorbic Acid Causes Copper Deficiency?… Huh?!?…

OK, where, oh where are we headed today?…
Some of you (actually several hundred of you…) saw and commented on my Cinco de Mayo MAG Post re the importance of using wholefood Vitamin-C COMPLEX and foregoing Ascorbic Acid…
And for those that may have missed it, here’s the permalink to that thread:
On many, many. many occassions, numerous MAG-pies have been asking for some “further proof” in the literature beyond the occasional reference to this notable “bait & switch”  that appears from time to time in alternative healing articles… Aside from the article embedded in that link above (Finley, et al Am Jrl Clin Nutrition, 1983), I happened upon an important find during my read & reflection this morning re this same topic…
For those that are really intrigued by this revelation re Ascorbic Acid and it’s impact upon Copper, a more pointed article is “Copper Nutrition During Infancy and Childhood,” by Lonnerdal, Am Jrl Clin Nutrition, 1998.
And just to expedite matters, I’m gonna copy an important set of paragraphs from pg 1050S of that article so that each of you can read from the horse’s mouth about HOW Ascorbic Acid CAUSES Copper Deficiency:
Ascorbic acid
Early studies on the effect of high concentrations of ascorbic
acid on copper absorption in experimental animals indicated that
copper absorption was reduced (71, 72). It was suggested that
this effect was achieved by a reduction of cupric (II) ions to
cuprous (I) ions and that the latter form is less well absorbed.
This interpretation, however, may be too simplistic; it is evident
that the interaction between copper and ascorbic acid is complex
and occurs at several levels. Ascorbic acid may have an effect on
copper at the absorptive stage as suggested by the earlier studies
and also some recent work in rats (73), but it may also affect
intracellular copper metabolism and transport of copper to the
liver, as well as copper excretion. In many studies it is difficult
to evaluate at which stage of copper metabolism ascorbic acid
interferes. The possibility of species differences in copper
metabolism should also be investigated as there appear to be differences
between results from studies in rats and human data.
Support for a so-called postabsorptive effect of ascorbic acid
on copper metabolism was provided in a study by DiSilvestro
and Harris (74). When copper-deficient chicks were injected
with ascorbic acid intraperitoneally, either in conjunction with
copper or 75 min before the copper dose, copper utilization was
markedly impaired. However, when ascorbic acid was injected
75 min after the copper dose, the activity of the copper-dependent
enzymes increased. The authors proposed that ascorbic acid
may be one of the reducing agents needed for the reduction of
ceruloplasmin-bound copper to make it available intracellularly.
It is also possible that ascorbate is needed for the transport of
copper from the mucosal cell to other tissues. Van den Berg and
Beynen (73) found that the major effect of high dietary intake of
ascorbic acid in rats was decreased copper absorption, but that
liver uptake and biliary excretion of copper also were increased.
The effect of ascorbic acid on copper metabolism was more pronounced
in copper-deficient than in copper-adequate rats. They
suggested that the high biliary excretion of copper was due to
increased liver copper uptake.

It is possible that the effect of ascorbic acid on copper metabolism
is less pronounced in human subjects than in animal models.
Finley and Cerklewski (75) found lower ceruloplasmin oxidase
activity and a tendency toward lower serum copper concentrations
after giving young, healthy volunteers 1500 mg ascorbic acid/d for
64 d. It is possible, however, that this was not due to decreased
copper absorption, because Jacob et al (76) found no effect on
copper absorption when ascorbic acid was given at different concentrations.
These investigators suggested that ascorbic acid can
cause release of copper from ceruloplasmin and thereby lower its
oxidase activity. This is a distinct possibility because immunologic
determination of ceruloplasmin showed no change in ceruloplasmin
protein concentrations. Feeding low-birth-weight infants
ascorbic acid–fortified formula (50 mg/d) did not result in any
negative effect on copper balance compared with feeding formula
with the normal amount of ascorbic acid (77).
I just wanted folks to have this more recent article, as well as related citations, for your reference and reflection. In a latter post this month, I’ll shed further light on WHY Copper deficiency is such a debilitating and destructive mineral dynamic in Homo Cupereidus…
As is often said on MAG, all is NOT as it seems…
Truth be known, I could write yet another book just about this article alone in terms of the implications that this dynamic of Ascorbic Acid has in lowering the bio-availability and viability of Copper in the cell, and the concomitant influx of Copper in the Liver that creates untold amounts of metabolic dysfunction… But more on that in a future post!
I just thought you all would like more definitive language and research findings to further embellish the earlier post this week on the Vitamin-C wars of the C-COMPLEX vs Ascorbic Acid, it’s synthetic and cheap impostor…
As always, I welcome your input, your questions and your insights… It would appear that the body of evidence that mineral deficiencies as a key mechanism for metabolic dysfunction grows larger and larger with each passing day!
A votre sante!
Citations for the Lonnerdal, 1998 AJCN Article cited above
71. Hill CH, Starcher B. Effect of reducing agents on copper deficiency
in the chick. J Nutr 1965;85:271–4.
72. Van Campen D, Gross E. Influence of ascorbic acid on the absorption
of copper by rats. J Nutr 1968;95:617–22.
73. Van den Berg GJ, Beynen AC. Influence of ascorbic acid supplementation
on copper metabolism in rats. Br J Nutr 1992;68:701–15.
74. DiSilvestro RA, Harris ED. A postabsorption effect of L-ascorbic
acid on copper metabolism in chicks. J Nutr 1981;111:1964–8.
75. Finley EB, Cerklewski FL. Influence of ascorbic acid supplementaton
on copper status in young adult men. Am J Clin Nutr
76. Jacob RA, Skala JR, Omaye ST, Turnlund JR. Effect of varying
ascorbic acid intakes on copper absorption and ceruloplasmin levels
of young men. J Nutr 1987;117:2109–15.
77. Stack T, Aggett PJ, Aitken E, Lloyd DJ. Routine L-ascorbic acid
supplementation does not alter iron, copper, and zinc balance in
low-birth-weight infants fed a cow’s milk formula. J Pediatr Gastroenterol
Nutr 1990;10:351–6.

Testing is Key to Hormone-D!

Testing is Key to Hormone-D!

Yes, this is what ALL the controversy is about with Hormone-D… getting from one state to the next!
The molecule on the Left is Calcidiol, aka 25(OH)D or the “Storage” form of the Hormone.
The molecule on the Right is Calcitriol, aka 1,25(OH)2 D3 or the “Active” form of the Hormone.
And you can’t get from Left state to Right state without consuming Magnesium and Mg-ATP… And that’s a fact.
Well, if you do nothing else today, please take the 10-min to watch this interview with Kenny L. De Meirleir, MD, PhD, a pre-eminent physiologist, internist and ME/CFS researcher based in Brussels:
He is MOST clear in his position re Hormone-D:

  • “Low 25(OH)D is just a ‘witness’ that you have too much 1,25(OH)2 D3…”  (this is ~4:30 in the video…)
  • He is critical of his professional colleagues for recommending so much “D”-pendence on this supplement…
  • He indicates the ratio of 1,25(OH)2 D3 (“Active”) to 25(OH)D (“Storage”) SHOULD BE 1.5, “maybe” 2 times greater, but what he is now finding are patients with FOUR TO FIVE TIMES (4-5X) more “Active” Hormone, compared to their “Storage” Hormone. It sets the stage for severe mineral and metabolic imbalance.

I have LOOOOONG suspected and theorized that this was, in fact, exactly what was happening, and now have clinical proof from one of the foremost authorities on this very dynamic. And why is this so alarming? Because HIGH levels of “Active” Hormone-D are indications of excess, unregulated Calcium in the blood. (And what makes it “unregulated?” — Too little Magnesium, as you no doubt imagined!…)
So, what to do?…

Please look BEFORE you “D”cide to supplement!
Conduct the following four blood tests to have certainty of your need for additional supplemental “D” — not because you read a glitzy article, or that your neighbor stressed the need, or worse yet, that your not-fully-informed doctor “ordered” you to do so. That they are choosing to overlook the metabolic foundation of this issue is a bit unsettling, at best…
The tests that get to the metabolic truth are as follows:

  • Magnesium RBC (Red Blood Cell): it’s the KEY catalyst for creating “Storage” and “Active” forms  of this Hormone…
  • 25(OH)D blood test: it’s the measure of the “Storage” form, the precursor to “Active” form of this Hormone…
  • 1,25(OH)2 D3 blood test: it’s the measure of the “Active” form of this Hormone…
  • “Ionized” Serum Calcium blood test (NOT a standard serum test!): given that Calcitriol’s JOB in the body is to put MORE Calcium into the blood stream, it only makes sense to know exactly how much you have there already, right?…

So, please, look BEFORE you take a “D”ive!
And if you need a refresher on why excess, unregulated Calcium is NOT your friend, please review this article that Carolyn Dean, MD, ND and I wrote in December, 2012:
I’m confident that this new information will be troubling to some, a bit confusing to others, but absolutely beneficial to all who take the time to read this, and act on it… I strongly encourage you to do so!
A votre sante!

The Truth of Hormone-D…

The Truth of Hormone-D…

Recently, one of the MAG-pies asked me about the “real” story on “Vitamin-D…”

Here are my unabashed comments to stimulate your neurons and hopefully nutritional sanity…
“Chances are you have very little valid information about your “true” mineral status… Serum measurements are NOT valid, as serum is “extracellular…” RBC measurements are QUITE valid (because they ARE Intracellular), but can get a bit pricey… HTMA (hair tissue mineral analysis) is QUITE VALID (it’s looking at tissue levels), but we are “conditioned” to question ANYTHING that is NOT BLOOD as a shaky source, and that is reinforced by practitioners who have been “TRAINED” to regard HAIR as “pure quackery,” which it is NOT.

“You have likely read hundreds of articles, and heard from ALL your family, friends, and work mates, telling you to take Vitamin-D, but what you did NOT know is that they failed to tell you the following important factors:
o Vitamin-D is a HORMONE…and a very powerful one at that…Despite it’s popularity, we as a species are NOT designed to EAT Hormones, but are, in fact, designed to make Hormones from Cholesterol… Hmmm… (You mean Cholesterol is actually good for my health?!?… Yeah, what a surprise!)
o Hormone-D MUST be in balance with Vitamin-A — it has been that way since the dawn of time… VERY FEW tell you that! Chris Masterjohn, PhD is a noted exception and authority here:
o That the conversion of Cholesterol >> Prehormone-D >> Calcidiol >> Calcitriol ALL require ENERGY (Mg-ATP) and Magnesium… Excess intake of “D” really drains your Maggie! (And does a number on your Potassium, btw…)
o That ALL 3 Hormones (Calcitonin, PTH and Hormone-D) that dictate levels and Location of Calcium are ALL activated by Mg… Imagine that… no one pushing “D” has stressed that, have they?…
o That Vitamin-K, the latest MSM (Main Stream Media) nutrient darling, MUST be Carboxylated and Phosphorylated to be biologically effective — fancy words for just another way to use up MORE Mg-ATP…
o That your doctor NEVER suggested you get 3 blood tests to accurately assess your need for Hormone “D”:
   1) Mag RBC (Ref Range: 5.0-7.0mg/dL — please totally ignore the current Ranges — OMg!)
   2) 25(OH)D — Storage Form (Calcidiol)
   3) 1,25(OH)2 D3 — Active Form (Calcitriol)
and if #1 is >6.0mg/dL AND #3 is LOW (please, just IGNORE #2…), then, and ONLY THEN. take a food-based form of Hormone-D, just like your Ancestors did for thousands of years… Yes, somehow they survived and you will, too… And a wonderful source is Cod Liver Oil that your grandparents, great-grandparents and their great-grandparents took…
o And yes, Hormone-D, the OLDEST HORMONE ON PLANET EARTH, is designed to get Calcium INTO the blood (Vitamin-A, btw, gets it into the Bone…) because the environment was very Mg-rich, and Calcium poor — and it was that way for millions of years until ~1900 when the Food Processors started to change it… And please know that excess dietary & supplementary Calcium is NOT your metabolic friend and the research is PROVING THAT IN SPADES (Bolland and Reid, 2009, 2010, 2011, 2012)… Trust me, there’s a reason why managing Calcium (which is Mg’s job in the body, btw…) is “Job #1” at BIG Pharma… Hey, we can’t rely on a mineral that CANNOT be patented, so let’s use Synthetic Rx Meds, that CAUSE Mg Loss, and then we’ll make billions!… (No doubt, you think I jest, I’m sure…)

“So, waaaaaaaay too much blah, blah, blah… but this topic is MY “Job #1″ to wake MAG-pies up to the metabolic reality of their body, and beat the drums of sanity in allowing the body to get back to metabolic balance…”

I’ll look forward to the blow-back and questions that this will no doubt stimulate…

A votre sante!

Giving Thanks!

Giving Thanks!

“If the only prayer you say is “Thank You…” it will be enough.”
— Meister Eckert (1260-1327)
Thanksgiving is, by far, my most favored of holidays! It’s a time we can all step off the hamster wheel for a day and truly reflect on the many, many blessings we effectively overlook the other 364 days of the year… It’s also a special time of fellowship with family, friends and community. Not to mention the great feast! — I was 50 before I realized how to enjoy this meal and not get lost in tryptophan overdose… And for those of the pigskin persuasion, it marks the initial stuffing of meaningful gridiron face-offs. And here’s the classic image of that first Thanksgiving that we can all relate to… I’ve always enjoyed this painting by Brownscombe which captures this cherished Colonial event, and even more so after I learned in my genealogical pursuits many years ago that the gentleman standing and offering “Thanks!” was, in fact, my 15th great-grandfather, William Brewster (1566-1644). Elder Brewster was born in Scrooby, Nottinghamshire, England, came over in the Mayflower and died in Plymouth, Mass. (btw, I descend from his daughter, Fear…)
And for those that want to learn the REAL story of what our ancestors did in Plymouth Colony, I would highly recommend Nathaniel Philbrick’s Mayflower ( You’ll never see this cherished holiday the same way again.
And all that said, you might say, that this feast of Thanksgiving is embedded in my genes!… Which leads me to my next point.
I’m about half-way through Bruce Lipton & Steve Bhaerman’s Spontaneous Evolution. (OMg, where’s he going with this one…) It’s really quite fascinating. The conventional thought out there is that ALL disease has a “genetic origin…”  That our genes just suddenly stop working correctly, stop producing life-affirming proteins & enzymes, and we are then left powerless because of the immutable nature of our cranky nucleotide pool. It’s a neat little theory, billions has been spent on trying to “prove” it (Human Genome Project), but it’s patently false and scientifically wrong. And the research to back that up that last statement is irrefutable, despite the growing acceptance that this Social Construction of Reality is increasingly being perceived as the Truth. Not so, grasshoppers…
The prevailing “scientific belief” is that DNA >> RNA >> Proteins >> our performance… That this is a one-way process and that our “Genes” dictate ALL, is very well understood and accepted, except by the scientists who are actually studying it closely. It just so happens that it’s a two-way process… that a lowly enzyme called “Reverse Transcriptase” enables the environment to shape gene activity and that a process called “Somatic Hypermutation” is triggered within to modify biological function to fit the changing environment. And good that these processes exist, as the entire process of Evolution depends on them!
And what triggers their activities and the myriad of functions that cascade from them? “Stress!”  
What a surprise!
And what nutrients are lost to “Stress!”?… Zinc, Magnesium and B-Vitamins… And what nutrients regulate and repair gene function and metabolism? Zinc, Magnesium and B-Vitamins… Hmmm… Anyone else seeing a pattern here?…
An intriguing thought, as noted in this book: “The caterpillar and the butterfly have the exact same DNA. They are the same organism but are receiving and responding to a different organizing signal.” And it turns out that our thoughts and beliefs have a profound impact on the performance and functionality of our physiology. As Bruce Lipton notes: “We see science climbing the proverbial mountain of knowledge only to find Buddha sitting at the top!”
So what does this all mean?… It’s time for us all to create a different “organizing signal…” Our bodies and our healing protocols dictate it.
And what’s the most powerful activator of the Parasympathetic Nervous System (the “rest and repair” component of the Autonomic Nervous System that mops up after the “fight or flight” response…)?
It turns out that more than the emotion of joy, more than the emotion of excitement, and even more than the emotion of love, the emotion of GRATITUDE lights up the brain and activates the Parasympathetic branch to work its MAG-ic. (Btw, this has been studied with PET Scanners and NMR Scanners.)
It turns out that the brain lights up like a Christmas Tree when we express “Thanks!” for our blessings…
So here’s a Thanksgiving tip guaranteed to create a “new” you… take a bath with Maggie this Thursday… Make it special,
and please enjoy the moment…
And about 1/2-way into the experience, close your eyes and give “Thanks!” to the Universe for your many blessings. Feel the sensation of “gratitude” in your heart and immerse yourself in this moment for a good 5 minutes. It will activate a shift in your being, and will create that much needed new “organizing signal” to enable healing changes within… Our clients that have done this have consistently let us know how grateful they were to have learned it…
So, I’ll leave you with that recommendation and I would like to close this post by thanking the many, many, many MAG-pies — around the world — for your participation in this evolving healing community. My cup, and bath!…, runneth over. I am blessed to be a part of this wonderful gaggle of folks, and just want to say, Thank you!, for making this a very special Thanksgiving…
Blessings to you all!
A votre sante!