Iron is the ‘stressor’ that is causing excess loss of magnesium. It is the causative agent to create oxygen stress, and nitrogen stress that is at the heart of neutering ceruloplasmin. Ceruloplasmin is the very protein/enzyme that is essential to properly manage this toxic metal and Morley talks about the studies on the structure and function of ceruloplasmin and the relationship with iron.
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Iron Toxicity Post #9: Bioavailable copper is essential to reduce iron-induced hydroxyl radical (*OH)!
Morley speaks on an article he found regarding the effects of iron overload on cardiomyocytes (heart) and how important ceruloplasmin is in managing that iron.
Iron Toxicity Post #8: Why Morley has ‘lost it’ and is obsessing over iron and the proper management of iron.
“When iron accumulates, chelatable iron replaces magnesium at the corresponding metal-binding site, promoting selective damage to these proteins.”
How 3,571 proteins require magnesium to work and how iron impacts on this during our lifetime.
Iron Toxicity Post #7: All manner of pathogens – fungal, viral, mycotoxin etc., MUST have iron to flourish and grow!
“All manner of pathogens – fungal, viral, mycotoxin, bacterial must have iron to flourish and grow”
Iron Toxicity Post #5: A deficiency of Ceruloplasmin (Cp) is one of the earliest manifestations of Copper deficiency
A deficiency of Ceruloplasmin (Cp) is one of the earliest manifestations of Copper deficiency.
Iron Toxicity Post #3: The underlying pathogenic event in oxidative stress is cellular iron mismanagement
The underlying pathogenic event in oxidative stress is cellular iron mismanagement and how is iron is managed inside our cells and bodies.