The object of Mother Nature is to mobilize iron, and thus, the importance of ceruloplasmin is to keep iron moving.
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Accumulation of iron in tissues (unbound) increases the risk of cancer.
Iron Toxicity Post #29: Irregular iron blood markers especially low ferritin is code for more magnesium, ceruloplasmin and B2
When iron blood markers show irregularities, especially low ferritin, we know that’s code for the need for more magnesium, ceruloplasmin (bioavailable copper) and riboflavin (B2).
The objective of iron metabolism is mobilization, and ceruloplasmin is the metabolic agent to guarantee that functional requirement is met.
Iron movement not iron storage as iron is supposed to RE-cycle. Find out why ferritin only is not the marker for anemia and the connection to ceruloplasmin.
Diabetes is not a medical disease; it is just copper and iron dysregulation.
Heart disease and neurodegenerative conditions and how unbound iron kills our mitochondria.
Iron Toxicity Post #17: Could ‘Folate deficiency’ be an EPI-genetic deficiency of bioavailable copper?
How folate deficiency and it relates to ceruloplasmin and epigenetics like MTHFR.
Iron Toxicity Post #11: If the Sun is the ‘center’ of our Universe, I’m coming to regard Ceruloplasmin as the ‘Sun’ of our universe of metabolic activity.
Iron is the ‘stressor’ that is causing excess loss of magnesium. It is the causative agent to create oxygen stress, and nitrogen stress that is at the heart of neutering ceruloplasmin. Ceruloplasmin is the very protein/enzyme that is essential to properly manage this toxic metal and Morley talks about the studies on the structure and function of ceruloplasmin and the relationship with iron.
Iron Toxicity Post #9: Bioavailable copper is essential to reduce iron-induced hydroxyl radical (*OH)!
Morley speaks on an article he found regarding the effects of iron overload on cardiomyocytes (heart) and how important ceruloplasmin is in managing that iron.