Iron Toxicity Post #38: Why Vitamin D and iron is not a good idea!

Iron Toxicity Post #38: Why Vitamin D and iron is not a good idea!

(Formerly #38)

This will serve as post #38 on Iron Toxicity and post #5 on Hormone-D.

In honor of everyone’s Independence Day, this simple post will set the record straight on why retinol (animal-based vitamin A) is so important. It is intended to address the two known forms of anemia plaguing this planet:

  • Low storage D
  • Low iron anemia

Both are cases of misinformation and are examples of how it is programmed, that are easily dispelled in the research literature if you were so inclined to find. 

As Mark Twain once said: 

“Whenever you find yourself on the side of the majority, it’s time to pause & reflect…”

Reflect we will, so let’s get started.  Please note that many people today are urged to take vitamin D by their healthcare practitioner.

The intake levels of vitamin D supplements are staggering: 5,000 IUs/day, 10,000 IUs/day, and 100,000 IUs/day etc.  The course record is a client in Buenes Aires whose doctor had him taking 250,000 IUs several times a week!  I believe that is outrageous insanity!

You will come to see how outrageous it is when you understand Mother Nature always provides that vitamin D with 10x more retinol which cod liver oil has the same unique and precise composition.  Cod liver oil is a rich source of the vital amine we call retinol. However, what’s most intriguing is the dark side to this ubiquitous vitamin D is it sensitizes the body to hydrogen peroxide (H2O2):

Weitsman, G.E., et.al. (2005). “Vitamin D sensitizes breast cancer cells to the action of H2O2: mitochondria as a convergence point in the death pathway.”

https://www.ncbi.nlm.nih.gov/pubmed/15964518

That is not a good thing, especially since excess unbound iron loves to interact with H2O2 and create the hydroxyl radical (OH*) through Fenton chemistry in the mitochondria. For those not sure what OH* radicals are, they are the most destructive chemical in the human body. In my humble opinion, they are the source agent for all the methylenetrahydrofolate reductase (MTHFR) madness that hijacks our metabolism and psyche.

The most notable symptom of ageing in our society today is the accelerated graying and the loss of hair on people, worldwide.  You have to wonder if there is possibly a connection to H2O2?

https://therootcauseprotocol.com/wp-content/uploads/2022/10/Reversing-the-Gray-Away-Naturally-38.pdf

After 7 years of meticulous research that article on “Reversing the Gray Away Naturally” is among the most important and revealing about what is really going on inside our toxic bodies. What is important to address is how it relates to oxidative process. While it is popular to think that the thyroid is the cause of the hair color changes or hair loss, I am totally not buying it and you should not either.

We need to look further than the toxic supplement of hormone D we may be taking in our regimen. One of our members, Kevin Kirkpatrick recently shared with us is his review of Chris Masterjohn, PhD’s work on retinol vs. vitamin D:

 

“Tufts University confirms that vitamin A protects against vitamin D toxicity by curbing excess production of vitamin K-dependent proteins.”

chrismasterjohnphd.com/2009/04/07/tufts-university-confirms-that-vitamin/

“…even modest amounts of vitamin D, whether provided by UV-light or in the diet, decrease liver stores of vitamin A; when the doses of D are larger, they decrease plasma levels of A as well. This suggests that vitamin D increases the need for and turnover of vitamin A.”

In other words, hormone D lowers retinol in the liver and the blood.  Hormone D increases the production and/or prevalence of H2O2. 

What’s equally as important is to understand that retinol (and the animal-based retinoids) has the ability to stop the H2O2 that ages us.

Mukherjee, S. et al. (2006).Retinoids in the treatment of skin aging: an overview of clinical efficacy and safety.”

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2699641/

Retinol is amazing stuff and I wondered if it might somehow be affecting our iron status?  Check our Chapter 5 (pg. 150-162) of this stunning book: 

Olson, J.A., Ross, A.C., (1996). “Vitamin A Deficiency.”

https://therootcauseprotocol.com/30330967_1661865995362modern_nutrition_in_health_and_diseases/

The opening paragraph said: 

“Anemia has long been recognized as a potential consequence of vitamin A deficiency.”

One of the known causes of iron anemia is lack of vitamin A, this means that those who are taking supplemental vitamin D are perpetuating their anemia status and not because of lack of iron! 

Again, I would reconsider whether to take supplemental vitamin D and act on Mark Twain’s wisdom instead: “Step back and reflect” on where you are and what you’re doing to challenge the societal trend of what is good for you.

We’re all about gaining health independence here at Magnesium Advocacy Group (MAG) and look forward to seeing our numbers continue to grow as more folks wake up to the toxic solutions being promoted by conventional practitioners all over this globe!

A votre sante!

MORLEY M. ROBBINS

For Facebook Discussion:

www.facebook.com/groups/MagnesiumAdvocacy/permalink/1059198477481554/

Iron Toxicity Post #37: There’s no such thing as heart failure!

Iron Toxicity Post #37: There’s no such thing as heart failure!

(Formerly #36)

I had no intention of posting so soon on the heels of #36, but the universe sees it differently. This article is simply too important not to share.

For anyone dealing with heart failure and edema, read this article consider asking that your cardiologist do the same:

Wang, Y., et al. (2011). “Iron-Induced Cardiac Damage: Role of Apoptosis and Deferasirox Intervention.”
https://pdfs.semanticscholar.org/dc73/5773862de2851dddf643275d9924e53d5583.pdf

“Excess tissue iron content as a result of iron overload or accumulation is estimated to affect up to 100 million people worldwide…Excess tissue iron accumulation is toxic and can lead to heart failure.”

There is no such thing as heart failure.  Note how the precision of how excess unbound iron activates the mechanisms of apoptosis and cell death. 

This is not only happening in heart cells (cardiomyocytes), this precise mechanism of cellular demise is occurring all over the body. Iron is attracted to areas where you hold emotional stress in your body. 

I encourage you to take the time to better understand how excess, unbound iron kills and how removing excess, unbound iron revitalizes our cellular machinery.

I am no fan of this synthetic iron chelator being promoted in this article. There are many proven natural mechanisms that have been proposed and discussed in earlier posts.

I’ve always wondered how my grandfather, Dada, died in his 93rd year. He had a very full life, but he suffered greatly at the end. And now I know the why, and the how, and hopefully so do you too.

A votre sante!

MORLEY M. ROBBINS

For Facebook Discussion:
www.facebook.com/groups/magnesiumadvocacy/permalink/1050501735017895/

Iron Toxicity Post #36: You are not anemic!

Iron Toxicity Post #36: You are not anemic!

(Formerly #35)

 

 

 

 

 

 

 

I would like to give some added traction to what I regarded as a very important post by Elizabeth yesterday:
www.facebook.com/groups/MagnesiumAdvocacy/1049485821786153/?notif_t=like_tagged&notif_id=1466274944603356

 

The ancient article (1973) that she posted is here:

Evans, J. L., Abraham, P.A. (1973). “Anemia, Iron Storage and Ceruloplasmin in Copper Nutrition in the Growing Rat.”


This more contemporary article (2014) was provided by Arne Kaminsky:

Manto, M. (2014). “Abnormal Copper Homeostasis: Mechanisms and Roles in Neurodegeneration.”
www.mdpi.com/2305-6304/2/2/327/htm

The point of this post is to underscore the importance of ceruloplasmin.  You are not anemic, rather you have low functional ceruloplasmin.

Let me share a recent client’s personal experience re this iron issue. What follows are set of blood panel markers, Pre- and Post-Cp Protocol:

  • Mag RBC (Old) 5.1 (New) 5.4
  • Ceruloplasmin (Old) 22.5 (New) 30.8
  • Serum Iron (Old) 55 (New) 150
  • Serum TIBC (Old) 285 (New) 334
  • % Saturation (Old) 19% (New) 45%
  • Serum Ferritin (Old) 53 (New) 52

What I want to make clear, anyone want to take iron supplements to correct their anemia; they should re-think their approach especially when you look at the rise of iron in the blood and the static level of ferritin.

Why I made the important point about Elizabeth’s post and Arne’s contribution is that ceruloplasmin (Cp) guarantees iron mobilisation. 

It ensures that iron is in active circulation around the body, just as Mother Nature intended it to be. This was very clearly understood up until the early 1980’s. A lot has happened since then.

What’s important to note is that, practitioners have an obsession over iron in storage, which is what ferritin tells us. Focusing on ferritin, stored iron, does not tell us what is going on with the active iron metabolism. 

By the time ferritin shows up in the blood, after it’s filled in the liver, the spleen and the bone marrow, it’s too late. Each ferritin molecule can hold up to 4,500 iron atoms, which is a major source for oxidative stress! And that’s what Sir Douglas B. Kell, PhD’s research is all about:

Kell, D.B. (2009). “Iron behaving badly: inappropriate iron chelation as a major contributor to the aetiology of vascular and other progressive inflammatory and degenerative diseases.” https://pubmed.ncbi.nlm.nih.gov/19133145/

Ferritin is pathophysiology, not a sign of vitality.

So with that in mind, let me share a series of key recommendations and please know that I am serious in this set of closing remarks:

  • Please get a full Monty Iron Panel to assess your true iron status:
    requestatest.com/mag-zinc-copper-panel-with-iron-panel-testing
  • Please get the panel interpreted by someone who knows what ceruloplasmin is and knows how important magnesium is to this process of iron metabolism.
  • Please pursue the Mother Nature Cp protocol for 3-6 months before re-testing.  Also before you take any more iron supplements to see if your iron shoots up too.

The way practitioners utilise ferritin only to access your iron status is not the way forward. It is like we do not allow a TSH only test for your thyroid.  It is time to demand a better understanding of what is really going on with your iron, especially since it is the base cause of oxidative stress. Out of control oxidative stress feeds all chronic diseases.

My obsession over iron is because iron eats magnesium for lunch.  The activation of 3,751 enzymes depends on magnesium as Mother Nature intends.

In my humble opinion, my understanding of anemia is that it is a clinical sign of inflammation and/or chronic disease. 

A votre sante!

MORLEY M. ROBBINS

For Facebook Discussion:
www.facebook.com/groups/MagnesiumAdvocacy/permalink/1050152505052818/

Iron Toxicity Post #35: Mother Nature intends iron to be moving not stored

Iron Toxicity Post #35: Mother Nature intends iron to be moving not stored

(Formerly #34)

Please say a prayer of thanks to Sir Douglas B. Kell, PhD (University of Manchester).

Kell, D.B., (2010). “Towards a unifying, systems biology understanding of large-scale cellular death and destruction caused by poorly liganded iron: Parkinson’s, Huntington’s, Alzheimer’s, prions, bactericides, chemical toxicology and others as examples.”
www.ncbi.nlm.nih.gov/pmc/articles/PMC2988997/pdf/204_2010_Article_577.pdf

Please share with your healthcare provider to highlight the dynamic between Fenton chemistry and ferritin.

Please take the time to read the highlights of this seminal study that flies in the face of convention. 

“I summarise the evidence that in a great many cases, one underlying mechanism, providing major stresses of this type, … of hydroxyl radicals via Fenton chemistry involving poorly liganded iron, leading to cell death via apoptosis..”

Mother Nature’s intention is to mobilize iron and ceruloplasmin’s importance is to keep the iron moving.

As we know there are practitioners that focus on storage of iron rather than what Sir Dr Kell is teaching us that iron is supposed to be moving around our body not stored. 

A votre sante!

MORLEY M. ROBBINS

For Facebook Discussion:
www.facebook.com/groups/MagnesiumAdvocacy/permalink/1035363813198354/

Iron Toxicity Post #34: Iron dysregulation causes cancer

Iron Toxicity Post #34: Iron dysregulation causes cancer

(Formerly #33)

Does anyone know the real metabolic origin of cancer?

Funauchi, Y., et al. (2015). “Regulation of iron homeostasis by the p53-ISCU pathway.”
www.ncbi.nlm.nih.gov/pmc/articles/PMC4642350/pdf/srep16497.pdf

Simply read the 1st sentence in the discussion section: 

“Recently, accumulating evidence has suggested a relationship between metal ions and cancer.”

The other metal issues noted are all caused by copper <> iron dysregulation and the corresponding lack of ceruloplasmin (Cp). This article, however, is silent on the role ceruloplasmin play.

It would be prudent for those who are worried about their chances of allowing cancerous metabolisms into their bodies had best get the Full Monty Blood panel. Then get it properly interpreted by a RCP practitioner who understands what iron anemia truly is. 

I hope this sheds new and important light on the relentless deception surrounding disease.

A votre sante!

MORLEY M. ROBBINS

For Facebook Discussion:
www.facebook.com/groups/MagnesiumAdvocacy/permalink/1030873433647392/