Low hemoglobin, ferritin, serum iron and saturation are all indications of iron dysregulation caused by low bioavailable copper which is expressed as low ceruloplasmin.
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Iron Toxicity Post #36: You are not anemic!
You are not anemic! Instead raise your ceruloplasmin because it guarantees iron circulation throughout your body.
Iron Toxicity Post #35: Mother Nature intends iron to be moving not stored
The object of Mother Nature is to mobilize iron, and thus, the importance of ceruloplasmin is to keep iron moving.
Iron Toxicity Post #34: Iron dysregulation causes cancer
Accumulation of iron in tissues (unbound) increases the risk of cancer.
Iron Toxicity Post #29: Irregular iron blood markers especially low ferritin is code for more magnesium, ceruloplasmin and B2
When iron blood markers show irregularities, especially low ferritin, we know that’s code for the need for more magnesium, ceruloplasmin (bioavailable copper) and riboflavin (B2).
Iron Toxicity Post #26: Red Blood Cell metabolism is by inference Iron metabolism
The objective of iron metabolism is mobilization, and ceruloplasmin is the metabolic agent to guarantee that functional requirement is met.
Iron Toxicity Post #25: Iron movement, not iron storage!
Iron movement not iron storage as iron is supposed to RE-cycle. Find out why ferritin only is not the marker for anemia and the connection to ceruloplasmin.
Iron Toxicity Post #24: Diabetes is NOT a medical disease!
Diabetes is not a medical disease; it is just copper and iron dysregulation.
Iron Toxicity Post #18: Most on this planet are dealing with ‘Subclinical Iron Overload’
Heart disease and neurodegenerative conditions and how unbound iron kills our mitochondria.
Iron Toxicity Post #17: Could ‘Folate deficiency’ be an EPI-genetic deficiency of bioavailable copper?
How folate deficiency and it relates to ceruloplasmin and epigenetics like MTHFR.