Hypoferremia occurs in any inflamed state and it is code for low bioavailable copper not lack of iron.
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How does iron get out of enterocytes? What causes buildup of iron in the tissues all over the body?
Iron Toxicity Post #71: There is no iron deficiency anemia on planet earth, but there is a pandemic of anemia adiponectin deficiency
There is no “Anemia” on Planet Earth, but there is a pandemic of anemia! adiponectin deficiency, a key and little discussed hormone whose dysfunctions are the gateway to insulin resistance, atherosclerosis, diabetes, hypertension, dyslipidemia, fatty acid dysregulation, PCOS, weight gain/obesity, increased inflammation, as well as increased oxidative stress.
Iron and the RBC (Red Blood Cells): Is this where the oxidative assault begins? [Hint: yes!]
Iron Toxicity Post #65: There is NO such thing as ‘Iron Metabolism’. There is ONLY Copper<>Iron Metabolism
This iron post has nine articles that examine iron metabolism and homeostasis. But nowhere do they highlight that there is no such thing as ‘Iron Metabolism’. There is only Copper<>Iron Metabolism, and Iron is the dummy!
Ferroxidase blocks the hydroxyl radical (OH*).
This post is based on a tale of two irons, hepcidin peptide that regulates them and causes chaos, confusion, clinical conditions and clinical currency.
Excess iron causes calcification all over the body, including the soft tissues and organs. We’re talking arterial plaque, cataracts, liver and gallbladder stones, etc.
Textbook by world-renowned hematologists gives us one sentence about the cause of iron overload and its impact on a litany of diseases. Absence of ceruloplasmin in the neurons and retina results in lipid peroxidation that in turn causes cell damage and death, followed by organ dysfunction.
Ferritin in the serum is a sign of tissue pathophysiology. Without the ceruloplasmin ferroxidase enzyme, iron doesn’t load properly into ferritin. So it gets damaged and takes on the properties of hemosiderin where iron is stored and doesn’t come out.